Preliminary research reveal conolidine may possibly inhibit specific ion channels, lowering neuronal excitability and restricting agony signals. This mechanism is particularly pertinent in neuropathic discomfort, wherever abnormal signaling results in persistent soreness. On top of that, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to agony notion. https://paulj516uab5.wikinewspaper.com/user