Gastrointestinal (GI): NSAIDs that block COX-one activity have correlations with GI complications mainly because of the inhibition with the synthesis of gastroprotective brokers including PGE2 as well as other prostaglandins. This inhibition brings about decreased mucin production by gastric epithelial cells, less bicarbonate secretion, and less epithelial cell turnover, among https://mahatmah109xbe1.angelinsblog.com/profile